Karin Knorr Cetina (page S76 ) cite the issue of consumer exploitation on anti ageing as a segment shrouded with an aura of science. “The use of human frailty for commercial exploitation is also singled out as a deplorable characteristic of the present-day anti-ageing medicines and market” as quoted from works tabled by McConnel & Turner.
1881 onwards Gerontology found itself being investigated concertedly by nations. (Juengst et al 2003) categorise mainly three parts or directions being followed by research work on ageing, mainly “compressed morbidity”, decelerated ageing” and “arrested ageing”.
Compressed Morbidity aims at preventing old age related disease, a paradigm relating to intervention in ageing processes at the molecular level to slow the process down. This desire or thought relates thus to life span increase. It is also the most fundamental or conventional one.
Decelerated ageing aims to slow down all fundamental cellular processes of the human body so as to raise the average life span and life expectancy levels overall.
Arrested ageing the most ambitious of all seeks to cure ageing by separating and doing away with all the remnants of damage caused by metabolic processes so as to be able to constantly maintain the vitality and bodily functions.
While the US and France have planned studies on ageing and further research on compressed morbidity approach, basic science has already established that life is a form of metabolism and has its own set of causes and effects, waste effects, lay byes.
These waste products accumulate to degenerate the form physiologically and biologically, slowly weaken and then die. These changes are the main course of research by modern life scientists with focus on how to control the damage caused by these side effects of life that cause ageing.
Mauron, Bruce, McConnel & Turner, in their study on anti aging make an observation on the intended life extension seeking projects that their consequence would result in “ life of the poor remaining 'short and brutish', while the rich could look forward to an extended enjoyment of their privileges.”
Authors such as Halldor Stefansson have argued that, contrary to the wear and tear of inanimate objects, aging in higher organisms is not primarily the result of damage to irreplaceable body parts. Certainly, molecules and cells can suffer from damage akin to wear and tear. Complex biological systems are dynamic and have the ability to repair and regenerate their damaged components. Even for components that cannot be replaced, like mammalian teeth, their degeneration can be seen not just as mechanical senescence but as limitations of the genetic program. There are differences in interpretation of aging changes which influence the way different researchers interpret the essence of aging; as discussed elsewhere, some authors see aging as genetic in nature while others see it as a build-up of damage counteracted by genetically-regulated mechanisms. Nonetheless, it is clear now that aging has a strong genetic component and it is not merely wear and tear.
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